"Ginkoum" txhais tau tias. Cov lus qhia rau kev siv thiab cov hauj lwm

Tshuaj "Ginkoum" phau ntawv qhia no qhia txog yuav ua li cas angioprotective txhais tau tias. Tshuaj yog ntawm zaub keeb kwm. Muaj ntau mistakenly ntseeg hais tias nws yog ib ntsiav tshuaj. "Ginkoum" yog muab nyob rau hauv daim ntawv ntawm cov tshuaj thiab qhov ncauj tov.

mechanism ntawm qhov kev txiav txim

Cov tshuaj muaj txhawb rau normalization ntawm metabolic dab nyob rau hauv lub hlwb thiab microcirculation, pab txoj kev rheological thaj chaw ntawm cov ntshav. Txhais tau tias, uas tswj cov kev ua ntawm lub vascular system. Lub mechanism ntawm qhov kev txiav txim yog ua raws li cov expansion ntawm me me cov hlab ntsha, ua rau kom venous laus. Cov kev npaj exhibits anti-edematous nyhuv los ntawm kev txo lub permeability ntawm cov hlab phab ntsa ntawm ob lub peripheral thiab cerebral.

Txhais tau hais tias "Ginkoum" (phau ntawv qhia no muaj cov ntaub ntawv xws li) tiv thaiv lub aggregation ntawm cov ntshav liab slows qhov kev txiav txim ntawm platelet activating tau. Nyob rau hauv thov medicament cim txhim kho nyob rau hauv cov ntshav mov piam thaj thiab oxygen mus rau lub hlwb.

Cov tshuaj niamntiav antithrombotic nyhuv. Cov nyhuv no yog manifested vim membrane stabilization nyob rau hauv cov ntshav liab thiab qe ua ntshav khov, normalization synthesis pg, txo cov kev ua si kev lom tebchaw thiab trombotsitoaktiviruyuschego tau. Cov tshuaj tiv thaiv tus tsim ntawm dawb radicals, peroxidation lipid oxidation nyob rau hauv cell phab ntsa.

Qhov zoo thaj chaw ntawm cov tshuaj "Ginkoum" phau ntawv qhia txog normalization tso tawm re-uptake thiab catabolism ntawm ib tug xov tooj ntawm neurotransmitters (acetylcholine, dopamine, norepinephrine), thiab lawv muaj peev xwm mus txuas rau lub receptors. Cov tshuaj tiv thaiv li ntawm cov fibrinolytic kev ua si ntawm cov ntshav, muaj anti-hypoxic nyhuv. Nyob rau lub hauv paus ntawm lub txais txhais tau tias nyob rau hauv lub hlwb tsub kom tsub zuj zuj makroenergov qab zib siv thiab cov pa normalized madiatornye Cns dab.

lub sij hawm

Tshuaj "Ginkoum" phau ntawv qhia xav kom vascular encephalopathy rau lub hauv paus ntawm lub taub hau raug mob, mob stroke rau cov laus uas muaj hnub nyoog. Cov tshuaj no yuav siv tau rau kev tshem tawm thiab nyem ntawm cov tsos mob xws li nco tsis tau thiab ib tug txo nyob rau hauv cov xim, pw tsaug zog ua cuam tshuam, kev ntxhov siab vim, deterioration ntawm kev txawj ntse muaj peev xwm. Tshuaj rau mob ntawm lub peripheral kev thiab microcirculation.

Los ntawm indications xws li Raynaud lub syndrome muaj xws li, arteriopathy lub sab nqua. Pom zoo tshuaj rau mob neurosensory hom manifested Gipoakuzija, tinnitus, kiv tauv hau. Los ntawm indications xws li ntshav qab zib retinopathy, macular degeneration (ua tem toob).

Contraindications

Tsis muab neeg sawv cev rau anticoagulation, ib tug neeg rhiab heev rau lub Cheebtsam infarction (mob). Cov tshuaj yog ua rau cov neeg mob uas ulcers nyob rau hauv lub mob huam, mob ntawm cerebral ncig mob cov hoob kawm, erosive gastritis. Tsis pom zoo kev kho mob thaum lub sij hawm cev xeeb tub, nyob rau hauv cov me nyuam, nyob rau hauv cov neeg mob uas arterial tawg. Txhob kho laus cov neeg mob.

"Ginkoum" txhais tau tias. Cov lus qhia. nqi

Cov tshuaj no yog pom zoo kom 40-50 milligrams los yog 1 p 3 ml / g. Duration ntawm kev kho - ib tug nruab nrab ntawm txog peb lub hlis. Tus nqi ntawm cov tshuaj - los ntawm 160 p.