Raws li tshuaj tua kab mob uas ua cov me nyuam

Ib tug ntawm cov feem ntau nyuaj lub sij hawm nyob rau hauv xyuas xyaum yog xaiv tus me ntsis tus neeg mob tshuaj tua kab mob. Tu siab, muaj ntau yam kab mob tshwm sim heev yooj yim, thiab tus me nyuam lub cev tsis yog ib txwm tau mus tiv nrog lawv nyob ib leeg. Lwm qhov teeb meem yog cov tsawg kev xaiv ntawm cov tshuaj thiab qhov fab ntxeev ntawm cov niam txiv.

Qhov ntawd yuav ua mob

Cov tshuaj tua kab mob yuav tsum tau muab xwb rau cov me nyuam tus kws kho mob. Cov tshuaj ua txawv, thiab nyob ntawm seb muaj hnub nyoog tau ob pab thiab raug mob. Cov me nyuam mos mus txog peb lub xyoos, txhob tshuaj "chloramphenicol" (vim lub high kev pheej hmoo ntawm central lub paj hlwb thiab lub siab puas) thiab "ceftriaxone" (haus yuav ua tau kom plab hnyuv colonization tom kom tsis muaj menyuam nws pathogenic muaj). Cov me nyuam nyob rau hauv 8 xyoo yog txhob aminoglycosides (muaj ib tug tshuaj lom ntxim rau cov hnov paj, muaj peev xwm tsim lag ntseg), tetracyclines (ua txhaum txoj kev loj hlob thiab kev loj hlob ntawm cov pob txha ntaub so ntswg, protein synthesis, kom lub siab).

Tshuaj tua kab mob rau cov me nyuam provokes cov tsos syndrome plab hnyuv dysbiosis. Raws li ib tug tshwm sim ntxhov qab los noj mov, quav, tshwm sim nyob rau hauv lub plab ntswj, xeev siab, tsam plab.

Yuav ua li cas yog cov kev cai koj yuav tsum paub txhua

Tshuaj tua kab mob rau cov me nyuam hais txog kev npaj uas yuav tsum tau noj nrog ceev faj. Kom tsis txhob tsis kaj siab txim koj yuav tsum paub cov nram qab no:

1. Tsis yog txhua txhua kab mob yuav tsum tau kho nrog tshuaj tua kab mob. Fungal thiab tus kab mob yog tsis zoo. Piv txwv li, nyob rau hauv tetanus, botulism, diphtheria.
2. Self-tshuaj yog tsis tau tso cai.
3. Muaj zog tshuaj cov kws kho tsuas yog rau cov kev indications. Qhov no yog vim lub fact tias tom qab ob los yog peb hnub ntawm thawj coj ntawm lub tshuaj tua kab mob miv nyuas siv zug yog txo vim muaj txoj kev loj hlob nyob rau hauv ib tug kab tsiaj txhuv resistant kab mob los ntawm cov kab mob thereto.
4. Child tshuaj tua kab mob (ncua kev kawm ntawv, ntsiav tshuaj, tsiav tshuaj, tshuaj) yuav tsum tau muab thiaj li maximize nws concentration nyob rau hauv lub lesion. Nyob rau hauv cov ntaub ntawv ntawm otitis media thiab ua pa kab mob nyob rau hauv kev xaiv li cas dropwise thiab aerosols. Tshuaj ntaub ntawv pom tau hais tias ib yam nkaus thiab lom thiab yog siv rau cov me nyuam xwb thaum tsim nyog kiag li. Niaj hnub nimno tshuaj muaj siab bioavailability thiab tso dag tso luag rau cov me nyuam organoleptic zog. Raws li ib qho piv txwv, tshuaj tua kab mob "Sumamed" rau cov me nyuam. Nws yog muag nyob rau hauv cov hmoov daim ntawv no, uas yog tsim los ntawm suspeziya. Lub tiav lawm khoom muaj ib tug ntxiag qaub ntxhiab thiab Sweet saj. Kua daim ntawv n nws feem ntau tsim nyog rau cov me nyuam me uas tsis tau nqos ib tug ntsiav tshuaj los yog cov tshuaj ntsiav.
5. Thaum xaiv ib tug tshuaj yog muab rau tus account rhiab heev rau lub pathogen. Vim lub fact tias cov kev ntsuam xyuas muaj peev xwm coj ob peb hnub, kev kho mob yuav tsum tau pib sai li sai tau, muab ib tug tshuaj tua kab mob rau cov me nyuam ntawm ib tug dav spectrum ntawm kev txiav txim.
6. Cov koob tshuaj tshuaj yuav yog nyob ntawm seb muaj hnub nyoog, tus kab mob, qhov hnyav, heev, hom pathogen.
7. Kom muaj qhov tsim nyog concentration ntawm cov tshuaj nyob rau hauv cov ntshav yog tsim nyog los soj ntsuam cov kev kho mob regimen. Hoob no ntawm kev kho mob yog nyob rau nruab nrab 7-10 hnub. Ntug lub sij hawm no yuav ua tau tsuas nyob rau hauv cov ntaub ntawv ntawm muaj mob loj.