Nce ntshav dawb

Nyob rau hauv tib neeg, muaj zog dawb ntshav hlwb - c yog dab tsi nws yuav tsum tau txuas? Ua lawv tus xov tooj nyob rau hauv cov ntshav ntawm cov neeg laus (ntau tshaj nyob rau hauv 9H10 cuaj hwj chim ib liter) yog hu ua leukocytosis. leukocyte theem nyob rau hauv cov ntshav ntawm tus me nyuam nyob rau lub hnub nyoog, muaj cov tshwj xeeb ntxhuav nrog xws li cov ntaub ntawv.

Muaj ntau ntau mechanisms rau txoj kev loj hlob ntawm leukocytosis:

• hematopoietic loj hlob ntawm cov kab mob vim yog lub transformation ntawm qia hlwb nrog ib tug polypeptide ntawm cancer;
• los ntawm tej yam ntuj tso yam stimulation leykopoeza xws li physicochemical yam inflammatory tshua tus me nyuam nyob rau hauv lub cev cov khoom cov ntaub so ntswg lwj, kev loj hlob ntawm hypoxia, qhov tshwm sim ntawm lub cev ceg, sib cais mus rau hauv cov ntshav uas muaj ntau yam co toxins, txhawb adrenal-pituitary system, accelerating tus maturation ntawm leukocytes nyob rau hauv hematopoietic tub ceev xwm;
• leukocytes nyob rau hauv cov ntshav nce muaj raws li zoo vim hais tias ntawm qhov kev tso tawm ntawm tus me nyuam ntawm ib tug loj tus naj npawb ntawm lawv nrog pronounced vascular tshua.

Ua lub xov tooj ntawm leukocytes yuav tsum yog physiological los yog pathological. Los ntawm pathological yog vim li cas vim hais tias cov neeg uas muaj txhawb cov ntshav dawb muaj xws li:

1. Ntau yam dab purulent thiab inflammatory kab mob xws li mob thiab subacute thiab mob cov ntaub ntawv.
2. Ntau pathological tej yam kev mob thiab cov kab mob uas ua rau cov ntaub so ntswg hypoxia, xws li hemolysis, hemorrhage, raug tshuaj lom tshuaj txuas hemoglobin.
3. Pathology, rau qhov tsim nyog ntawm cov uas yog necrosis ntawm ntaub so ntswg, xws li plawv nres tej hauv nruab nrog cev, cwj nrag, kub nyhiab rau loj cheeb tsam ntawm daim tawv nqaij thiab qog ua kua week.
4. Kab mob ntawm noninflammatory cim, nrog mob hnyav qaug cawv, xws li ntshav qab zib ketoacidosis, uremia thiab cov zoo li.
5. Lom ntau tej yam tshuaj lom.
6. Phem onkoobrazovany.
7. Vim lub loj hlob ntawm hematopoietic kab leukocytes nce tshwm sim nyob rau hauv cov kab mob xws li erythremia, mob thiab ntev leukemia, polycythemia, thiab lwm tus neeg.
8. Cov kab mob thiab tej yam kev mob uas nrog tiv thaiv kab mob tshuaj, xws li ntshiab kab mob, ntau yam collagen, glomerulonephritis, thiab cov zoo li.

leukocytes kuj nce thaum raug physiological yam tseem ceeb xws li:

• noj cov zaub mov nplua nuj nyob rau hauv proteins;
• tseem ceeb lub cev tom;
• muaj zog psycho-kev nyuaj siab;
• overheating;
• hypothermia;
• thaum lub sij hawm cev xeeb tub.

Qhov kawg ntawm lub physiological yog vim li cas, vim hais tias ntawm cov uas muaj peev xwm yuav nce ntshav dawb, xam tau tias yog nyias. Cev xeeb tub poj niam ua ntej tus me nyuam ntawm analyses muaj kom tsis txhob muaj kev nyuaj siab thiab lub cev tom kom deb li deb li sai tau kom tsis txhob muaj kev siv tshuaj, yuav tsum tsis txhob tau txais yog kub dhau heev lawm los yog ib yam nkaus thiab txias da, vim tag nrho cov ntawm cov yam tseem ceeb los ua ib tug es muaj zog hloov mus hloov los nyob rau hauv cov ntshav leukocytes. Thiab yog hais tias nyob rau hauv lub qub xeev, xws hloov mus hloov los yuav tau kav liam, ces lub cev xeeb tub tus kws kho mob yuav tsum tau nqa tawm ib tug sib sib zog nqus kev soj ntsuam ntawm lub cev ntawm cov poj niam. Tiam sis tej kev kho mob manipulation, txawm lub feem ntau txawm - nws yog ib qho ntxiv kev nyuaj siab rau lub expectant niam thiab cov me nyuam hauv plab. Vim hais tias, cia li xav hais tias, vim hais tias nws yuav tsim nyog los tshem tawm cov daim ntawv loj loj uas tau pathologies xws li:

1. Ntau yam kab mob - ob qho tag nrho cov kab mob thiab kab mob, fungal, protozoal (meningitis, otitis, tonsillitis, mob hlab ntsws thiab ntau lwm tus neeg).
2. Lub xub ntiag ntawm mob thiab ntev inflammatory dab nyob rau hauv lub cev xeeb tub.
3. Aggravation ntawm ntev-kho thiab tsis nco qab lawm mob tus kab mob uas yuav inevitably tshwm sim raws li ib tug tshwm sim ntawm ib tug weakening ntawm kev tiv thaiv thaum lub sij hawm cev xeeb tub.
4. Txawv localization ntawm purulent dab.
5. Txhua yam ntawm cov kab mob ntawm lub urinary system.
6. Cov kev tsis haum ntawm tej etiology.
7. Lub xub ntiag ntawm cua nab.
8. Tej kev fab loj intoxication.
9. Lub cev qhuav dej ntawm phem onkoobrazovaniya.
10. Mob pancreatitis, peritonitis los yog mob hnyuv tws.
11. Stroke.
12. Hemorrhage ntawm ntau yam localization.