Yuav ua li cas yog sab laug ventricular diastolic kawg hom 1?

Diastolic kawg ntawm sab laug ventricle 1 hom - dab tsi yog tus kab mob no thiab yuav ua li cas nws yuav tsum tau kho li cas? Lo lus teb rau ib lo lus nug, peb mam li muab cov ntaub ntawv yog muab los ntawm tsab xov xwm. Nyob rau hauv tas li ntawd, koj yuav kawm tau yog vim li cas xws ib qho txawv txav tshwm sim, thiab yuav ua li cas cuab kev pom ntawm nws muaj peev xwm qhia tau.

lus qhia dav dav

Peb yuav tsum nrhiav tau tawm yog dab tsi no lub cev ua ntej teb cov lus nug ntawm yog vim li cas muaj yog tshuav ventricular diastolic kawg hom 1.

Lub sab laug ventricle yog hu ua ib tug ntawm cov 4-eh lag hauv lub plawv ntawm tus txiv neej. Hais tias nws originates kev (voj voog loj), uas muab ib tug nruam ndlwg ntawm cov ntshav nyob rau hauv lub cev.

Yuav ua li cas hom ntawm tus kab mob?

Diastolic kawg hais yog ib feem ntawm lub plawv yog hu ua ib tug tseem ceeb yuav txo tau nyob rau hauv nws muaj peev xwm los txij ntshav nyob rau hauv cov kab noj hniav ntawm ib tug pulmonary leeg. Nyob rau hauv lwm yam lus, xws li ib tug pathological mob ua rau nws tsis yooj yim sua kom zoo ncig.

Yog li, sab laug ventricular diastolic kawg hom 1 yog ib tug loj heev lub plawv kab mob, uas yog yus muaj los ntawm ib tug tsis muaj lub peev xwm ntawm hais tias lub cev feem mus so kom txaus thaum lub sij hawm diastole. Nws yuav tsum tau muab sau tseg hais tias nws yuav siv sij hawm kwv yees li ntawm 0.4 vib nas this. Lub sij hawm no yog heev txaus siab rov qab kho cov laus thiab lub zog ntawm lub plawv nqaij fullness.

Lub txaus ntshai tus kab mob?

Poob ventricular diastolic kawg tshwm sim los ntawm hom 1 drop ncaws feem, uas ntxiv ua rau ib tug cim txo nyob rau hauv stroke volume. Yuav kom tiv thaiv stagnation ntawm cov ntshav nyob rau hauv ob lub ntsws thiab pab kom txawm peem lub dilation pib nce nyob rau hauv systolic ventricular volume. Yog hais tias xws tiv thaiv cov tshuaj tiv thaiv ntawm tus kab mob yog tsis ua raws li, nws zoo nkaus li meej kev hem thawj pulmonary tawg (retransmission), thiab ho tsub kom lub load rau ntawm lwm yam ventricle (txoj cai), uas tom qab ua rau ib tug txo ntawm nws volume. Raws li ib tug tshwm sim, lub diastolic siab tej zaum nws yuav venous congestion. Yog hais tias muaj yog ib tug loj heev kawg, nws yog ib qho yooj yim los mus tsim koj muaj teebmeem kev edema.

Tej zaum yog vim li cas

Yog vim li cas muaj peev xwm tsim sab laug ventricular diastolic kawg hom 1? Yog vim li cas rau qhov no phenomenon pw nyob rau hauv lub nram qab no:

• hypertensive tus kab mob;
• hypertrophic cardiomyopathy ;
• infiltrative (periodic), kab mob plawv (ie, lub plawv yuav nres, coronary artery kab mob, mob tawg (arterial) thiab hypertrophy ntawm tus neeg mob cov kab txiav uas yog nyob sab nraum lub cheeb tsam ntawm dilation thiab thinning).

tej yam tshwm sim ntawm rejection

Xws li ib tug mob feem ntau ua rau txoj kev loj hlob ntawm ob pulmonary arterial thiab venous tawg. Qhov no pathological mob tej zaum yuav manifest nws tus kheej raws li nram no:

• pheej hnoos (feem ntau paroxysmal);
• nocturnal dyspnea (paroxysmal);
• ua tsis taus pa.

Yuav ua li cas lwm cwj pwm hais tseg sab laug ventricular diastolic kawg hom 1? Cov tsos mob ntawm xws li ib tug loj tus naj npawb ntawm variations yuav hawm tsis manifest nws tus kheej. Txawm li cas los, nrog cov kev loj hlob ntawm tus kab mob, cov neeg mob yuav pib kom raug cai nyob rau hauv xws nta li:

• kev mob nyob rau hauv lub plawv, uas yog paroxysmal nyob rau hauv cov xwm (raws li nyob rau hauv coronary artery kab mob);
• o ntawm lub qis extremities;
• ua tsis taus pa (tej zaum yuav tshwm sim txawm nyob rau hauv so);
• spasticity;
• koj luv luv ua tsis taus pa.

Thaum xws li cov tsos mob yuav tsum nco ntsoov sab laj ib tug kws kho mob. Tom qab tag nrho, lub ntxov ib tug pathological mob yog kuaj, qhov yooj yim dua nws yog coj kev tswj. Yog hais tias koj xa cov kab mob no yog kuaj lig dhau lawm, hais tias nws kho mob yuav kav rau ib tug ntev heev lub sij hawm, nrog rau daim ntawv thov ntawm loj nyiaj ntawm cov tshuaj thiab tag nrho cov tsim nyog cov txheej txheem.

Yuav ua li cas kho tau?

Tam sim no, ib tug tib kiag uas yuav tau raug lees paub tias los ntawm feem ntau cov kws txawj, yog tsis muaj. Qhov no yog tej vim lub fact tias tus kab mob yog phem txaus rau kev tshawb nrhiav. Raws li hais saum toj no, xws txawv tshwm sim rau ib tug ntev heev lub sij hawm, asymptomatic, uas ua rau tus neeg mob thov kev pab kho mob lig dhau lawm.

Yog li ntawd yog dab tsi yog tias koj pom tawm sab laug ventricular diastolic kawg hom 1? Kev kho mob ntawm tus kab mob no yog los tshem tawm qhov ua rau uas ntxias tsis zoo tiam sis. Yog li, cov neeg mob yuav tsum:

• kho uas twb muaj lawm ischemia;
• Koj normalization ntawm lub plawv dhia;
• txo cov ntshav siab.

Nyob rau hauv tas li ntawd, raws li qhov kev nrhiav kom tau ntawm xws li ib tug pathological lub xeev ntawm ib tug neeg mob prescribers pab pawg neeg ACE blocker. Feem ntau cov feem ntau cov kev xaiv ntog rau cov tub txawg 'Lizonopril ". Nws muab nyob rau hauv cov ntsiav tshuaj daim ntawv rau 20-40 milligrams ib hnub (nyob rau hauv ob kauj ruam).

Zoo tau nyob rau hauv cov kev kho mob ntawm qhov no txawv yuav ua tau tiav, thiab los ntawm kev siv ntawm calcium blockers. Yog li, ob pab pawg ntawm cov tshuaj txo cov ntshav siab, ho thiaj li plawv cov ntaub so ntswg oxygen thov, raws li tau zoo raws li nres thiab txo hypertrophy ntawm sab laug ventricle. Incidentally, vim lub txais tos ntawm cov tshuaj yog zoo diastolic mob ua hauj lwm, uas ntxiv ua rau normalize hemodynamics.

Qhov zoo tshaj plaws kev tshwm sim muaj cai nyob rau hauv kev kho xws tus kab mob thaum ua ke nrog mob-sparing diuretic tshuaj. Thaum tsim nyog kiag li tej zaum yuav siv thiab lwm yam antihypertensive tshuaj.