Noj qab haus huvCov kab mob thiab mob

Gout: Kev Kho Mob tshuaj. gout

Gout tshwm sim nyob rau hauv yuam cai ntawm purine metabolism hauv tib neeg lub cev. Thaum zoo li no yog kuaj nyob rau hauv cov ntshav uric acid concentration yog inflated, thiab ob leeg thiab / los yog periarticular ntaub so ntswg, ob lub raum thiab lwm yam kabmob cuam tshuam los ntawm urate deposits - sodium urate ntsev.

Cov ua rau gout

Lub tshaj uric acid yog tsim nyob rau hauv tus muaj zog synthesis ntawm endogenous purine txo feem ntawm urate los yog thaum lawv tseem xaiv dab. Paub cov tsos mob ntawm thawj thiab tus kab mob. Cov yav tas rooj plaub yuam ua hauj lwm them sai sai mus tshuaj npaum li cas los ib tug txiv neej uas raug ntaus los ntawm gout. Cov ua rau tus kab mob yuav tsum tau ncauj lus kom ntxaws saib xyuas.

Gout, uric acid overproduction provoked

Oversupply ntawm purine bases thiab nucleosides - lub ntsiab yog vim li cas rau lub siab concentration ntawm uric acid. Nrog ib tug unlimited zaub mov noj, nplua mias uas muaj substrates txoj kev purines, lawm tshwm sim hyperproduction acid.

Cov tsos mob theem nrab ua hyperproduction excessively siab rhuav tshem cov hlwb tshwm sim nyob rau hauv ib tug tom qab ntawm tej yam kab mob (provocateur, piv txwv li, yuav Hematological malignancies paraproteinemia, hemolysis, alcoholism, anticancer kws khomob). Hyperuricemia feem ntau nrog psoriasis. Txawm li cas los, tus soj ntsuam pathology ntawm gout muaj nyob rau hauv cov neeg mob feem ntau.

Gout yog tshwm sim los ntawm txo feem ntawm uric acid

Nyob rau hauv 90% ntawm cov neeg uas ris lub nra nrog tus kab mob no gout, muaj yog txo acid extraction. Cia lub cev pab uric acid raum, lub plab thiab daim tawv nqaij. Los ntawm kev txo qhov feem ntawm urate los ntawm ob lub raum, lawv yog nyob rau hauv abundance noog nyob rau hauv lub cev thiab crystallize. Me me quav muaju tso nyob rau hauv cov pob qij txha, uas ua rau o thiab mob. Ib tug raum urate thaum ciav jade.

Ib tug zoo xws li cov phenomenon tej zaum ua rau diuretics, haus dej cawv, acetylsalicylic acid, aminophylline, caffeine, diazepam, diphenhydramine, L-dopa, dopamine, vitamins B 12 thiab C, nyob rau hauv me me noj tshuaj npaum cas. Lead ua rau phaum mob kab mob. Lead gout ua xim surrogate cawv thiab lwm qhov chaw uas muaj hnyav hlau.

Symptomatology gout: cov tsos mob chua leeg

Tshwm sim ntawm cov tsos mob txuam nrog ntsev deposits tsim uric acid. Lub muaju tso nyob rau hauv cov pob qij txha thiab ob lub raum. Tus thawj nres ntawm gout feem ntau tsis tshwm sim ua ntej lub hnub nyoog ntawm peb caug. Nws kev tshaj cov neeg mob mus txog 40-60 xyoo.

Tus nres yog nrog los ntawm o thiab liab ntawm cov pob qij txha, mob morbidity. Yog tsis muaj kev kho mob, nws tsis kis tau rau ob peb hnub los yog txawm lub lis piam. Yav tsaus ntuj thiab hmo ntuj - feem ntau ntawm cov sij hawm ntawm tshwm sim ntawm nraum tawm tsam.

Provoked gout tshaj nqaij khoom noj khoom haus, haus dej haus cawv, tej yam qib ntawm ntses, kas fes thiab lwm yam khoom enriched nrog purine bases. Tus kab mob yog nyob rau hauv feem ntau tus neeg mob muaj feem xyuam rau tus ntiv taw xoo, tab sis lwm yam pob qij txha yeej yog tsis cais. Nws ciav cov tsos mob yog mob mob caj dab, uas tshwm sim thaum ib tug phiv raum pathologies.

mob gout

Arthritis - mob hauv daim ntawv ntawm gout. Nws yog qhia nyob rau hauv lub cia li yuav los tom ntej gouty nres - ib tug mob o ntawm cov pob qij txha. Thaum gouty mob caj dab urate muaju poob tawm ntawm cov ntaub so ntswg nyob rau hauv cov pob qij kab noj hniav, ua o. Gouty tawm tsam tshwm sim, feem ntau yog thaum lub sij hawm hmo ntuj.

Provoked los ntawm dej cawv, kev poob plig, lub cev overload, kev phais, tom ntej no gout tshuaj. tshuaj kho mob muaj peev xwm tshem tawm tawm tsam thiab tsis tus mob. Thaum zuj txha thiab lwm tus ntaub so ntswg swell, ntawm daim tawv nqaij ntawm qhov chaw ntawm lub hearth blushes, mob ua.

Mob caj dab tawm tsam ploj nyob rau hauv ib ob peb hnub (tej zaum lub lis piam), txawm yog hais tias lawv yog tsis kho. Ua tawm tsam rov tom qab rau lub hlis los yog ib xyoos. Txawm tias twj ywm sij hawm thiab tej zaum ntev li 10-20 xyoo. Cov neeg uas tsis baffling kev kho mob, cov zaus kev tawm tsam yog nce, uas yog muab kev koom tes nyob rau hauv ntau yam pob qij txha. Lub caij ntawm sib cem nce, lawv ua ntau pronounced.

mob gout

Mob kab mob no yog los ntawm cov yam ntxwv nta:

  • mob mob caj dab;
  • txuam nrog ntawm urate muaju;
  • raum kab mob.

Ntev gout yog nrog los ntawm tag nrho cov cim ntawm lub sij hawm. Mob caj dab cuam tshuam cov neeg uas nyob ntawm lub qhov hauv daim ntawv ntawm tus kab mob no, ncua los yog tua kab mob kho tau.

Nrog lub yeej ntawm ob lub raum muaj 3 hom hloov:

  1. Raum urate muaju overgrown nrog cov ntaub so ntswg. Lawv ntws weakly qhia inflammatory cov tshuaj tiv thaiv.
  2. Intrarenal txoj kev khiav ntawm cov zis urate muaju daig.
  3. Cov mob txeeb zig yog tsim uric acid pob zeb.

Modifications ua ke nyob rau hauv txawv variations, uas ua nyob rau hauv lub soj ntsuam lub sij hawm "gouty raum". Lub pob zeb zalegshie nyob rau hauv lub mob txeeb zig, yog asymptomatic los yog muaj ib co yam ntxwv ntawm urolithiasis.

Txoj kev kho mob

Qhov teeb meem ntawm kev kho mob yog los mus tiv thaiv gouty tawm tsam thiab tshem ntawm mob mob caj dab. Txhawb kom txaus dej kom tsawg, nyhav dhau heev lawm pib dua, noj cov zaub mov txoj kev kho, kev siv yeeb tshuaj yuav txo tau, ua hyperuricemia, gout tiv thaiv tsuas yog haus dej cawv. tshuaj kho mob muaj peev xwm tshem tawm o thiab mob.

Dej thiaj li concentration ntawm urate, tsis pub lawv poob mus rau lub muaju tso nyob rau hauv cov ntaub so ntswg thiab kabmob. Nyob rau hauv tas li ntawd, nws tswj lawv leaching thiab tshem tawm ntawm lub cev. Haus dej cawv muaj peev xwm sawv ntawm diuretic nyhuv. Nyob rau hauv loj koob tshuaj nws dehydrates lub cev, yooj yim lub crystallization ntawm ntsev pib gouty nres. Ntxiv mus, nws slows cia lub feem ntawm urate los ntawm lub cev, uas lawv yuav tsum tau tso rau hauv cov pob qij txha.

Poob ceeb thawj yuav pab txo txoj kev pheej hmoo ntawm recurrent gout tawm tsam. Cov khoom noj yuav tsum txo roj thiab txo calorie ntau ntau. Noj cov zaub mov ua ke nrog aerobic ce ua.

tshuaj kho

Main yam uas tswj gout, - kev kho mob nrog cov tshuaj, pab mob, eliminates ib tug inflammatory teb, tswj cuam tshuam metabolism ua rau hyperuricemia. Tshuaj loog yog ua nrog cov tshuaj acetaminophen (Tylenol) los yog los ntawm lwm yam ntau haib analgesics.

Ntawm nonsteroidal anti-inflammatory siv yeeb tshuaj indomethacin pom zoo. Txawm li cas los, nws tsis yog haum rau cov neeg mob uas muaj kev tsis haum rau cov tshuaj aspirin nyob rau hauv cov neeg mob uas qhov ntswg polyps. Rau kev tshem tawm ntawm gouty nres yog tsim colchicine.

Cov neeg mob cov lus siab rau uas nrog qhov ncauj tshuaj muaj kev phiv loj heev uas txuam nrog rau zaus ntawm nws txais tos (txhua txhua teev los yog ob teev mus txog rau thaum tus mob no tsis yog ho khiav rov qab los sis tsis tshwm sim cov nyom tshua).

Corticosteroid teej tug mus rau cov haib anti-inflammatory kab mob, pab mob tawm tsam. Kom tsis txhob mob loj ntev-lub sij hawm tshwm sim los ntawm cov tshuaj luv luv kev kawm. Lawv yog muab rau cov neeg mob muaj concomitant cov kab mob ntawm daim siab thiab ob lub raum.

Ntev sij hawm cov neeg mob noj tshuaj uas txo lub concentration ntawm uric acid nyob rau hauv cov ntshav. Lawv pab mus rau txoj kev sib nrauj ntawm hnyav tofusnyh deposits tiv thaiv pob zeb tsim thiab kev loj hlob ntawm lub pathology nyob rau hauv lub raum. Ua tsaug rau lawv, yog tsis muaj re-gout. tshuaj kho mob ntawm pawg no los yog tsub kom lub feem ntawm urate, los yog thiaj li lawv synthesis ntawm purines zaub mov.

Cov tshuaj yuav paug arthritic nres, li ntawd, lawv txais tos yuav pib tom qab lub attenuation ntawm lub inflammatory txheej txheem. Yog hais tias lawv twb kho ua ntej xwb, cov hoob kawm tsis cuam tshuam, thiab sim kho lub tshuaj (tshwj xeeb tshaj yog tom qab nws extinction). Txij li thaum muaj ntau cov neeg mob uas muaj ntau ntau cov uric acid pob zeb tsim yog tsis raug nyob rau hauv ob lub raum thiab tsis tab kaum cov gout, kev kho mob nrog cov tshuaj uas cuam tshuam rau theem ntawm urate yog purely ib tug neeg ua cim.

Nrog sulfinpirazona probenecid thiab txhim khu lub feem ntawm uric acid. Cov tshuaj tsis haum rau cov kev kho mob ntawm cov neeg uas muaj urolithiasis. Lawv tej zaum ntxias tsim ntawm lub pob zeb. Hnyav haus, ua ke nrog lawv kom tsawg, txhawb ceev los ntawm zaj acid urinary system thiab tiv thaiv cov tsim ntawm conglomerates.

Vim allopurinol tiv thaiv uric acid synthesis. Lawv inhibit qhov kev metabolic hloov dua siab tshiab ntawm purine bases rau hauv lub acid. Nws ua tib zoo tshuaj vim hais tias cov kev pheej hmoo ntawm kev phiv rau cov neeg uas tau lus zoo raum muaj nuj nqi.

Siv hauv tsev

Tsev kho mob pab txo cov tsos mob ntawm gout, tam sim no sharply. Inflamed ob leeg yog raug tsa sawv, muab nws so. Morbidity yog txo los ntawm thov dej khov. Cais txais tos aspirinsoderzhaschih tshuaj (lawv inhibit qhov kev feem ntawm uric acid).

Kho tshuaj ntsuab: rosehip thiab cov hiav txwv buckthorn, cranberries thiab txiv pos nphuab, thiab blueberries Rowan, barberry, thiab St. John lub wort, birch thiab txiv qaub, chicory thiab lwm yam tshuaj ntsuab. Siv homeopathic tshuaj.

Noj cov zaub mov txoj kev kho rau gout

Dieting kuas ruaj khov so ib pliag. Qhov tseeb tiag mas, mus cuag cov tag nrho tshem tawm ntawm cov khoom enriched nyob rau hauv purines. Yog hais tias tus tsis muaj kev zam tsis yooj yim sua mus kom ib tug tshaj plaws tsis pub tshaj rau lawv noj. Sim sib npaug rau tus nqi ntawm cov roj thiab tshaj haus.

Lub tshaj nyeem purines anchovies, sardines, gus, nqaij qaib thiab tej roj nqaij, offal, nqaij cia, taum thiab cawu cub. Thaum tus mob ntawm gout txiv lws suav noj nyob rau hauv moderation.

Lub soob yog tag nrho ntawm cov organic acids. Purines nyob rau hauv lawv tsis yog li ntawd ntau npaum li cas raws li, piv txwv li, nyob rau hauv cov nqaij. Yog li ntawd, nutritionists cov neeg uas yog quaj los kuamuag vim gout, noj txiv lws suav tsis txwv tsis pub. On qhov tsis tooj, lawv ntseeg hais tias ib tug tsim nyog tus nqi ntawm txiv lws suav thiab tais diav yog pab tau rau cov neeg mob.

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