Lyme borreliosis: etiology, cov tsos mob thiab kev kho mob

Lyme borreliosis yog tshwm sim los ntawm spirochetes, thiab yog kis tau los ntawm zuam. Nws yog ib hom kab mob yus muaj los ntawm ib tus mob kawg. Feem ntau cov cuam tshuam daim tawv nqaij, licas, lub paj hlwb thiab lub plawv. Rau cov thawj lub sij hawm zuam-borne Lyme kab mob sau npe nyob rau hauv lub teb chaws USA (txiv qaub), li ntawd, tus kab mob no yog hu tej zaum ua - Lyme kab mob.

Lyme borreliosis: etiology.

Tus kab mob ua rau spirochetes teej tug mus rau lub genus Borrelia. Intermediate fwjchim luj kawg nkaus yog zuam. Reservoir borreliosis yog cov tsiaj qus thiab nas. Heev feem ntau, muaj ib tug mixed kab mob nrog zuam-borne encephalitis thiab Lyme kab mob.

Zuam mov Lyme kab mob: cov tsos mob.

Tus kab mob tau sau npe rau txhua sab av loj tsuas yog Antarctica. Rau teb chaw xws li Russia Leningrad, Kaliningrad, Tyumen, Yaroslavl, Perm, Tver, Kostroma cheeb tsam, raws li zoo raws li lub Far East, cov Urals thiab West Siberian cheeb tsam. Nws yog recalled hais tias nyob rau hauv lub Leningrad cheeb tsam zuam-borne Lyme kab mob no yog kis thiab European taiga hav zoov zuam. Xwv vectors yuav tsum yog los ntawm 5 mus rau 90%. Lub ncu tus kab mob lub sij hawm ntawm tus kab mob yog hais txog 14 hnub. Early soj ntsuam kos npe rau ntawm tus kab mob no yog liab (yaug kais dej) ntawm daim tawv nqaij ntawm qhov chaw ntawm lub tom ntawm Ixodes zuam. yaug kais dej ntawm daim tawv nqaij yog kho kom zoo, reddened chaw yuav ntsuas mus txog rau 10 centimeters nyob rau hauv txoj kab uas hla. Account mob qhov twg no cov nqaij ntawd mus txog 60 cm los sis ntau tshaj. Thaum lub sij hawm, qhov chaw ntawm lub chaw puv dawb thiab yuav xiavlus (cyanotic) tinge tsim ib lub nplhaib zoo. Lub tom kiav txhab yog tsim thiab ces tus rumen. Qhov no qhov chaw yog khaws cia rau 14-20 hnub thiab ces disappears. 30-45 hnub tom qab ntawm cai soj ntsuam cov tsos mob ntawm sab hauv nruab nrog cev kev koom tes.

Zuam mov Lyme kab mob: pathogenesis.

Thaum ib tug raug tus kab mob zuam tom tas Lyme kab mob no yog ib tug kab mob. qaub ncaug Borrelia tau rau hauv daim tawv nqaij, thiab ces nyob rau hauv ib ob peb hnub ntawm intensive yug me nyuam rau, kis lwm qhov chaw ntawm lub dermis thiab cov hauv nruab nrog cev (daim siab, lub plawv, cov pob qij txha, lub hlwb). Lub causative tus neeg saib xyuas rau ib tug ntev lub sij hawm yuav laus nyob rau hauv tib neeg lub cev thaum ua mob tus kab mob.

Lyme borreliosis: mob.

Yuav kom paub meej tias cov mob (Lyme disease) yog tsim nyog los ua ib txoj kev kawm ntawm cov ntshav. Kuaj thiab kev kho mob ntawm tus kab mob no yog nyuab los ntawm qhov tseeb hais tias tus sau los ntawm tus neeg mob kev kho mob keeb kwm ntaub ntawv tsis qhia qhov tseeb ntawm lub tom. Yog li ntawd tus neeg mob tsis tau txais kom tsim nyog kev kho mob, uas yuav ua tau kom xiam oob qhab. Serological kev ntsuam xyuas nqa tawm siv enzyme immunoassay. Neurological ces thiab xyuas cov leeg nrob kua.

Lyme borreliosis: kev kho mob.

Rau cov kev kho mob ntawm borreliosis xaiv causal thiab pathogenetic txoj kev kho. Nyob rau hauv thawj zaug rau theem ntawm tus kab mob muab tshuaj tua kab mob. Tso dej tshuaj tua kab mob (tetracycline kab) nyob rau hauv lub siab tshaj plaws koob tshuaj muab txaus ntau nyob rau hauv hlwb txha nqaj qaum kua. Kev siv cov tshuaj tua kab mob nyob rau hauv thaum ntxov ua sawv tiv thaiv txoj kev loj hlob ntawm mob, thiab neurological teeb meem artralgicheskih. Cutaneous ces borreliosis nrog tetracycline doxycycline nyob rau hauv ib lub hlis. Thaum txhab ntawm poob siab system ntawm ampicillin, ceftriaxone, cefazolin, roxithromycin, cephalosporin, azithromycin, erythromycin. Nyob rau hauv cov pob qij txha zoo kho nyhuv yog tau thaum uas siv cov tshuaj anti-inflammatory siv tshuaj (naproksin, hlotazol, indomethacin, plakvinil), analgesics thiab physiotherapy.